The Best Chiropractic Care in Mesa, Arizona
by James Bogash
CRP, Metabolic Syndrome, and Risk of Cardiovascular Events
While the rest of the medical community is debating whether we should check CRP levels in patients, these authors decided more than eight years ago that is was worth checking. While the the results of this study are no big surprise, we do see that CRP values are important markers for CVD risk in patients with Syndrome X.
Circulation — Abstracts: Ridker et al. 107 (3): 391
by James Bogash
Effects of a Dietary Portfolio of Cholesterol-Lowering Foods, vs Lovastatin on Serum Lipids and CRP
The pharmaceutical companies are scrambling to find a drug that lowers CRP levels. Well–look no further–Mother Nature has provided us with the tools already.
In this study we find that a mixed dietary approach at lowering cholesterol and CRP was as effective as lovastatin. I’m not being cynical here–I truly do wonder why a study like this published in JAMA does not get any press. Can the pharmaceutical influence be so powerful as to suppress this kind of info from the general public???
JAMA — Abstracts: Jenkins et al. 290 (4): 502
by James Bogash
Insulin resistance and CRP as independent risk factors for non-alcoholic fatty liver disease
I just thought I would use this article to tie in with the new documentary “Supersize Me.” This is exactly what happened to the star of this film. He ended up with elevated liver enzymes just 3 weeks into his program. The fast food had induced insulin resistance and elevated inflammation in this short a period of time.
Insulin resistance and C-reactive protein as independent risk factors for non-alcoholic fatty liver disease in non-obese Asia.
by James Bogash
Increased CRP in Polycystic Ovaries: Marker of Cardiovascular Disease
This is another good example of the intricate web that is physiology. While change is occurring but slow, many ob/gyns will treat PCOS with the birth control pill, which will worsening insulin sensitivity. If treated this way, the physician is missing the boat entirely, and will leave the physiological imbalance we know as Syndrome X to run rampant and destroy the patient’s health.
JCEM — Abstracts: Boulman et al. 89 (5): 2160 –
by James Bogash
Predisposition to Atherosclerosis by Infections
This article adds further weight to the contribution of infections to cardiovascular disease. There is a growing list of factors that contribute to risk, and many, if not most, can be managed with lifestyle changes…CRP, cholesterol, homocysteine, HDL cholesterol and now infections. Maintaining a lifestyle that supports healthy immune function (avoiding refined carbs, lots of fruits and veggies..) can help lower risk of infections and possibly lower CVD risk.
Circulation — Abstracts: Prasad et al. 106 (2): 184
by James Bogash
Emerging Cardiovascular Risk Factors
Okay…the research has been out for many years. Long enough for medical journals to be writing summaries. So why are they not becoming the standard of care? Currently, if someone is at risk for heart disease (strong family history, sedentary, overweight) all that is typically run is a lipids panel (cholesterol, triglycerides). As a patient, you need to insist on more important tests as well…homocysteine, CRP, fibrinogen…
Clin Rev Spring:33-38, 2000 Nearly 50 years ago, epidemiologists coined the term “risk factors” to characterize personal traits and living habits that indicate an increased probability of cardiovascular disease (CVD). This simplistic concept provided the means to develop strategies for preventing CVD and its complications. Recently, the term “primordial prevention” has been used to indicate prevention of the modifiable risk factors. Some indicators of risk, including age, male sex, low socioeconomic status, and family history of premature CVD, are not modifiable. For other factors, intervention has been proven to lower CVD risk or is likely to lower CVD risk. Although control or elimination of established risk factors for CVD can markedly reduce the likelihood of coronary heart disease (CHD), approximately half of all patients who have had a CHD event have no major risk factors. …many potential new markers of CHD are under investigation. It is o! ur purpose to review those emerging risk factors relevant to plaque vulnerability — markers of inflammation, infection, an atherogenic/prothrombotic state, and genetic factors. Elucidation of new markers provides possible insights into the mechanisms of atherogenesis. In time, these emerging risk factors may lead to further explanation of CHD events and new interventions.
by James Bogash
Dietary fatty acids affect plasma markers of inflammation
I find it ironic that for decades, mainstream medicine did not think that diet had much to do with the disease process. And the idea that a diet could be “pro” or “anti-inflammatory” was just short of malpractice.
This article looks into the different pro-inflammatory effects of several types of fat and their effect on markers of inflammation like fibrinogen and CRP. As expected, trans and saturated fats had a negative effect. Stearic acid, which is found in milk chocolate and cocoa butter, did raise levels of fibrinogen.
AJCN — Abstracts: Baer et al. 79 (6): 969 –
by James Bogash
CRP and Other Circulating Markers of Inflammation in the Prediction of Coronary Heart Disease
Articles like these, if they get to the mainstream press (and I did see this one in one of the Phoenix major newspapers) will just add confusion. Basically, CRP is a risk factor for CHD. No question.
This study evaluated the available data and determined that the risk factor for higher CRP levels in about 1.45 times the risk. The authors strongly downplay this risk. However, they also note that cigarette smoking increases risk 1.87 times and elevated cholesteol 2.35 times. Considering how easy it is to lower CRP levels through dietary means, and considering that lowering overall inflammation will probably lower you risk of other chronic diseases, I cannot see why we would not consider CRP in our workup of CHD.
NEJM — C-Reactive Protein and Other Circulating Markers of Inflammation in the Prediction of Coronary Heart Disease.
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