The Best Chiropractic Care in Mesa, Arizona
by James Bogash
Alcohol Consumption and Plasma Concentration of CRP
Looking for ways to lower CRP levels? Moderate alcohol consumption was shown to lower CRP levels in this study. This article did not differentiate between types of alcohol intake, but my guess would be that red wine intake would affect CRP levels the most.
Circulation — Abstracts: Albert et al. 107 (3): 443
by James Bogash
Comparison of CRP and LDL in Prediction of First Cardiovascular Events
I find it amazing, with today’s technology, that this article is no longer news!! I’m sure everyone has heard about this article already. What does amaze me, is that discussion in the medical literature still does not think that CRP levels should be checked routinely. My gosh…do we have to stamp “CRP” on a bat and whomp these doctors upside the head so they see it every-time they look in the mirror?? Personally, I’ve been telling people for years that CRP levels are important for assessing CVD risk (along with homocysteine, Lipoprotein A, ApoE4, fibrinogen…). However, I did have a gentleman in a class I was teaching today say that his cardiologist did not feel a need to check CRP levels because it was already known that he had CVD. HUH? Maybe the cardiologist did not order it because he/she would not know how to lower it if it was elevated? What if this patient’s CRP levels are elevated and interventions such as avoiding refined carbs and fish oil can bring it down? I guess we’ll never know.
NEJM — Abstracts: Ridker et al. 347 (20): 1557
by James Bogash
Incidence of High-Strain Patterns in Human Coronary Arteries–Assessment With Three-Dimensional Intravascular Palpography and Correlation With Clinical Presentation
I thought this was interesting. We are starting to get pretty ingenious with our cardiovascular imaging (and opening up cans of worms in regards to exactly how well we are really “treating” CVD). With this type of ultrasound imaging the physician can actually tell if the plaque is deformable or not. The deformable plaques are much more likely to rupture and lead to heart attacks. Interestingly, these plaques also correlated with CRP levels.
Circulation — Abstracts: Schaar et al. 109 (22): 2716 –
by James Bogash
Dietary Fiber and C-Reactive Protein
I’ve said it before…why all the ruckus trying to find a drug that will lower CRP levels? We already have many natural approaches that will lower CRP levels–exericse, avoiding refined carbs, fish oils… Many clinicians are still not ordering CRP levels for their patients at high risk of heart disease. As far as I’m concerned, the evidence is very strong that CRP is a risk factor for many chronic diseases. I always run CRP levels in my office anytime I requiest bloodwork.
nutrition.org — Abstracts: Ajani et al. 134 (5): 1181 –
by James Bogash
CRP, Homocysteine Related to Frequent Fruit and Vegetable Intake
All this fuss trying to find a drug to lower CRP levels. It’s almost laughable. And you know when they find that some drug (most likely front runner is the statins) lowers CRP it’ll be all over the news with recommendations to have CRP levels checked and drugs used to manage levels.
Nutrition.org — Abstracts: Gao et al. 134 (4): 913 –
by James Bogash
Effect of physical activity on serum C-reactive protein
I find it interesting that this study was funded in part by Bristol-Myers-Squib, a drug company. That aside, there are currently ongoing trials looking for drugs to lower CRP levels. We have a medical establishment not willing to fully admit the impact of chronic inflammation on many aspects of health, but yet trials are underway to lower CRP levels with drugs. The cynic in me would suggest that, as soon as a drug is developed to lower CRP, the editorials will come piling in suggesting that inflammation is a major causative factor in CVD. Exercise and lowering refined carb intake will both easily and safely lower CRP levels already. How bout shifting all that money for R & D somewhere more useful?
Cardiosource –
by James Bogash
Hyperhomocysteinemia as independent risk factor for silent brain infarction
I recently had a patient who had asked her primary care doctor to run a homocysteine level based on some stuff she had been reading in layman’s journals and her family history. Her doctor refused. I have routinely been running homocysteine and CRP levels on patients for quite awhile now. To say that elevated homocysteine (whether by itself or indirectly as a marker for altered folate/B12 status) has an impact on our health would be an incredible understatement. I am going to make a pretty strong statement here…any clinician who refuses to run a homocysteine level when a patient not only has a family history but REQUESTS the test should no longer be practicing. Being a physician and having patient’s health in your hands requires certain committments–one of those would be to crack a medical journal at least once a month.
Neurology — Abstracts: Kim et al. 61 (11): 1595 –
by James Bogash
Magnesium supplement intake and C-reactive protein levels in adults
Given that inflammation is a major player in most, if not all, chronic diseases, anything that can lower inflammation is going to have a positive effect on our risk of chronic disease.
This study finds that patients with low dietary intake of magnesium, those taking magnesium supplementation had lower CRP levels, a marker of inflammation. I usually don’t recommend magnesium separately (except in cases of constipation, in which it can work very well), but a good quality multi should have the magnesium needed when coupled with a good quality diet.
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