LifeCare Chiropractic

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Gastroenterology

Short-Chain Fatty Acids Induce Intestinal Epithelial Heat Shock Protein – (11-05-01)

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Short-Chain Fatty Acids Induce Intestinal Epithelial Heat Shock Protein

I know this is one of those long, complicated titles, but the take home message here is clear. SCFAs definitely protect the lining of the GI tract (this is a rat study but there have been many such studies in humans). To get SCFAs, we need a combination of both healthy bacterial flora and soluble fiber.

Gastroenterology — Abstracts: REN et al. 121 (3): 631

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Intestinal dysfunction, liver cirrhosis & spontaneous bacterial peritonitis – (06-25-01)

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Intestinal dysfunction, liver cirrhosis & spontaneous bacterial peritonitis

I know it’s a big long title, but basically, patients with liver cirrhosis have increased suseptability to bacterial infections without any known source of the bacteria. This article suggests that the bacteria may come from the gut. This would suggest that the use of probiotics in any patient with liver disease would be an essential part of their therapy.

Synergy : Journal of Gastroenterology and Hepatology 16 (6), 607-612

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Hypochlorhydria, Inflammation, Parietal and G-Cell Populations – (01-10-02)

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Hypochlorhydria, Inflammation, Parietal and G-Cell Populations

This study has incredible implications. This is a mouse study, but if it holds true in humans (and if this concept ever makes it out of this journal…) it could blow a hole in one of the highest prescription classes in Western civilization.

This article suggests that the normal stomach up-regulates its ability to make MORE acid in response to inflammation caused by bacterial infection. On the other hand, stomachs with impaired gastric acid production (hypochlorhydria), are more susceptible to infection and further injury from bacteria. The hypochlorhydria can be genetic or from treatment with acid suppressive drugs. The bottom line–we need to seriously re-evaluate or use of acid suppressive therapy in patients with gastritis and ulcers and especially H. pylori infection.

Gastroenterology — Abstracts: ZAVROS et al. 122 (1): 119

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